SARS2 pandemic gets crazy as climate-changed 2020 ends
FPMag has surveyed many doctors around the world and there seems to be one main consensus, “We are getting tired.”
Experts are not too concerned about the recent genomic anomaly in Great Britain. It is somewhat of a tempest in a teapot when prevention effort should be the primary public focus. Locking out Britain was a fool’s game. Locking out the virus is what is needed.
Obviously the vaccines now coming available are promising against the SARS-CoV-2 virus. At least two of those approved are MRNA vaccines which are based on a synthetic replication of the SARS2 and hence can be modified if ever that were needed, say experts.
The variant in Britain is still the same basic SARS2 and no expert involved has said that the approved vaccines will not be effective against anomalies within the virus, in fact the WHO has issued a statement saying that in the affirmative. Getting the doses into arms is a daunting task, however, and relief is still far off on the horizon say many experts.
by Micheal John
There is much talk in the general media about the alleged virus from Britain, but among doctors it is only moderately interesting, just another modification within a very stable genome that has thousands of little quirks.
Taxonomically, SARS-CoV-2 is just another strain of the severe acute respiratory syndrome coronavirus family (SARSr-CoV) having genetic similarity to a genome already found in bats.
Humans should not be catching this virus. We know it is induced mostly into the nose when we breathe and hence we should be protecting each of our respiratory systems and preventing the distributed exhalation by infected persons.
“There are many thousands of lineages of SARS-CoV-2 which differ on average by only a small number of defining mutations. It remains true that SARS-CoV-2 currently in global circulation have little genomic diversity. Subtleties in the mutations carried in different lineages can, however, be very useful for reconstructing patterns of transmission.” Citing Lucy van Dorp, Senior Research Fellow, Microbial Genomics, UCL
Showing 1211 of 3568 genomes sampled between Sep 2020 and Dec 2020.
Click any image to enlarge and read.
B.1.1.7 (VUI–202012/01) is not the menace. Catching the coronavirus is the menace.
Some research indicates the very specific British ‘B’ variant (B.1.1.7) may have developed in the body of an immunodeficient patient for months. The lingering presence allowed the virus time to “learn” (it doesn’t have a brain but it is engineered for not only survival but also proliferation) more about the human body. The B.1.1.7 has 23 unique characteristics, some of which are enablers for transmission.
Doctors are keen on how to treat patients and save lives.
“That doesn’t mean it is more harmful, it simply means there is now even greater reinforcement for following the health guidelines for preventing infection,” says Dr. Sanjiv Hyoju, a clinician and scientist presently at the University of Chicago but a principal epidemiologist in Nepal. Dr. Hyoju is one of the doctors FPMag has been talking to about the treatment of COVID-19 using available and proven pharmacological solutions. He and colleagues recently wrote in a yet-to-be-reviewed study report (SSRN-id3743118)
“Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a life-threatening viral infection that can rapidly spread and be lethal. Although many patients with mild symptoms recover, an acute form of the infection is characterized by rapidly evolving respiratory failure, an acute inflammatory response, organ failure, and death. As a mechanism to intervene in the acute inflammatory consequences of this infection, here we present three cases of moderate to severely symptomatic SARS-CoV-2 patients recovered following clonidine treatment. In this limited report, we proposed several mechanisms by which clonidine may be useful in managing rapidly evolving SARS-CoV2 infection based on the rationale that early clonidine administration can intervene in the neuromodulatory response that characterizes rapid patient heath deterioration in this COVID-19 infection in a vulnerable population.” Citing study report: Mitigation of Symptomatic SARS-CoV-2 with Clonidine: A Case Series from Nepal. ‘A Neuromodulatory Approach to Prevent Complication and Death’
A Changing Virus is Expected and perhaps more of them as Earth’s Climate Changes and challenged species shed their viral loads.
In fact the relationship between respiratory disease and climate change is well known according to dozens of studies.
We also know that the virus behaves differently on reinfection of previously infected persons and that, as in the case of minks in Europe, when the virus transmits to an animal and again later back to a human, it has undergone a number of worrisome changes. The humans and other mammals they live within the same dramatically changing climate, are challenged. And now they are shedding and sharing homicidal pathogens.
There is a strong argument in that coincidence to suggest that “Climate Change” is the bigger topic
COVID-19, the story of the year is indeed a threatening disease growing more troublesome as global deaths approach 2 million and cases soar in a nearly vertical climb.
Can humans afford to ignore any longer, what is causing the proliferation of bat (CoV) and monkey (Ebola) pathogens in humans?
The SARS-CoV-2 virus lived as similar genomic variants within bats for anywhere up to 50 million years. An old, stable virus, it learned our bodies. Deny it access.
“A 2017 study of 12,333 bats from Latin America, Africa, and Asia found that almost 9% carried at least one of 91 distinct coronaviruses. The authors estimated that there are at least 3200 coronaviruses that infect bats. Moreover, there are over 1400 species of bat. Figuring out which ones are susceptible to which coronaviruses is no small task. Bats are incredibly varied and successful creatures. In evolutionary terms, fruit-eating bats diverged from insect-eating bats some 50 million years ago.” Citing “The Lancet: The origin of SARS-CoV-2“
The bat has much to teach us, apparently. Bats have suppressed coronaviruses for millions of years. What is it about their immune system that we can borrow? Bats are mammals just like humans. There are many similarities. FPMag has been talking to numerous experts about this for the last couple of years. There is widespread interest in the scientific community, especially among epidemiologists. There is something in this field totally amazing, yet to be known. Hopefully 2021 will open more doors.
Some bats are experiencing immune system craziness with a new fungus spawned by climate change, similar to what humans are experiencing from Coronaviruses: our immune systems go crazy causing wild storms of overreaction.
Bats’ immune systems go berserk like ours do on many occasions when encountering something new, like the white nose syndrome, a fungus-caused disease among bats in North America.
Like the SARS-CoV-2 is to humans, the white-nose fungus is new to bats. Caused by the rise in temperature of the Earth (Climate Change), the multi-million year old immune system of the many species of bats impacted, is stymied. Their hibernation is interrupted; their immune systems go ‘crazy’ like ours do when we encounter a heavy viral load of SARS2; and they get quite ill, shedding their viruses in their caves like a snotty-nosed kid.
Background reading on this topic:
- Climate Change: Replace leaders who pollute. Hold a family meeting. Here we go…
- Is Climate Change Real?
- G20: Deal with Climate Change. End wars. What about Jamal?
- What do Climate, Idlib and COVID19 have in common? The ugly, the bad and the good.
- Yes. Wear a mask. Wear a NIOSH N95 & be climate conscious.
- EU a competent polycentric player. Processing change in response to American threat.
- How To: Be climate friendly. Love Macron’s G7 priority, Earth.
- My Doctor Fauci. Video Insights, CoVID19 pandemic. Here we go, back to school to learn biology.
- People not government can defeat SARS2
SARS2 Update 2022-01-20 01:51 GMT
- 253 Regions reported 339,035,374 cases
- 56,775,974 cases active
- 5,601,277 people reported killed by COVID-19
- 1.65% is current Case Fatality Rate (CFR)
- 276,658,123 survived COVID-19
Beta Technology Global Estimates
- 22.10% of all humans (1,739,251,468) have been infected
- 0.74% Global estimated inferred average Infection Fatality Rate (IFR)
(influenza is .1% or 6 per 100k (2019))
- 12,821,042 Total deaths (estimated actual) including errors, and unreported likely-cause excess deaths such as people who never went to a hospital but had COVID-19 indications but never tested.
EPICENTER: USA (68,686,441)
- 105.99% of the USA may have been infected or even reinfected including reported + estimated unreported mild and estimated asymptomatic (352,361,442.33) persons, some of whom may not have been ill in their first course of the disease, but could have spread the disease.
- 1.27% is USA current Case Fatality Rate (CFR) &
- 0.28% is estimated inferred average Infection Fatality Rate (IFR)
- 986,612 estimated total COVID-19 deaths including unreported likely-cause excess deaths. According to projections of IHME, IHME calculation of excess deaths is higher than what CSPaC is showing.